Sure, it keeps staff safe, but all kinds of drugs can do that...while inducing drowsiness and offering the minimized awareness we associate with comfortable sedation in ourselves.Ace, by contrast, doesn’t necessarily achieve this.
Consider Thorazine (chlorpromazine): This drool-on-yourself, One Flew Over the Cuckoo’s Nest kind of tranquilizer was once ubiquitous in human psych wards. Problem is, not only do we have the case of dysphoria and Thorazine to consider, we also happen to know that acepromazine causes even more profound dysphoria in humans than “Vitamin T” does. You may abhor the comparison between humans and animals in this case.
Though this acepromazine-like drug is still used, it’s not exactly a kind drug when used in big, human-stopping doses. After all, humans and animals don’t react the same way to all kinds of drugs.
Again, if animals are like humans, the tranquilization acepromazine offers allows for continued awareness (perhaps even heightened awareness).
In fact, that’s technically the difference between a tranquilizer and a sedative.
That’s why, I believe, acepromazine continues to hold sway in small animal veterinary medicine for sedation.
That, its low abuse potential, its relative safety, its effectiveness..the price factor, of course.
I once actually witnessed the death of a cardiomyopathic Doberman who struggled and went into ventricular fibrillation while being restrained for an EKG; the owner watched the whole thing!
While there is always an exception to the rule, appropriate sedation is my preference for handling these patients, hands down! before radiographs, IV catheter, thoracocentesis, echocardiogram, etc.
I have a great deal of experience with this situation, and strong convictions as well.